Thesis on viruses

This procedure contradicts many modern medical viewpoints. Gallstones are thought to be formed in the gallbladder, not the liver. They are thought to be few, not thousands. They are not thought to be linked to pains other than gallbladder attacks. It is easy to understand why this is thought: by the time you have acute pain attacks, some stones are in the gallbladder, are big enough and sufficiently calcified to see on x-ray, and have caused inflammation there. When the gallbladder is removed the acute attacks are gone, but the bursitis and other pains and digestive problems remain.

Hypovirulent phenotypes do not appear to correlate with specific genome features and it seems there is not one particular metabolic pathway causing hypovirulence but several. [39] In addition to negative effects, beneficial interactions do also occur. Well described examples are the killer phenotypes in yeasts [40] and Ustilago . [41] Killer isolates secrete proteins that are toxic to sensitive cells of the same or closely related species while the producing cells themselves are immune. Most of these toxins degrade the cell membrane. [40] There are potentially interesting applications of killer isolates in medicine, food industry, and agriculture. [20] [40] A three-part system involving a mycovirus of an endophytic fungus ( Curvularia protuberata ) of the grass Dichanthelium lanuginosum has been described, which provides a thermal tolerance to the plant, enabling it to inhabit adverse environmental niches. [42]

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Thesis on viruses

thesis on viruses

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